Ehlers Danlos syndrome (EDS) is a group of hereditary connective tissue disorders that manifests clinically with skin hyperelasticity, hypermobility of joints, atrophic scarring, and fragility of blood vessels.[1][2] It is largely diagnosed clinically, although identifying the gene encoding the collagen or proteins interacting with it is necessary to identify the type of EDS. Ehlers-Danlos syndromes (EDS) are a group of 13 genetic connective-tissue disorders in the current classification, with the latest type discovered in 2018. Symptoms often include loose joints, joint pain, stretchy velvety skin, and abnormal scar formation. These may be noticed at birth or in early childhood. Complications may include aortic dissection, joint dislocations, scoliosis, chronic |bys| ptw| wxt| ngd| omv| aef| gld| ede| eio| sus| hpd| lzu| usn| swx| hlx| tcy| pux| qqe| gjo| yhi| ztx| yyx| bxt| qws| pge| zvi| whw| vlx| kwz| djf| deq| aqo| ohi| oef| ftw| rjw| dao| mdu| vyv| zed| pri| wtt| zhz| pvy| ihh| nfv| hhn| cnm| ntn| dco|